This is basically what the "embalmers found blood clots" people actually believe about vaccines.
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@Drain Todger in a nutshell.
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Those calamari clots that they're pulling out of dead people are giant hunks of amyloid and fibrin, or "fibrinaloid". They've actually stained them and determined their composition.
The “clots” featured in these videos are not blood at all; they are primarily amyloid proteins (thanks to analysis done by Mike Adams of clots supplied by Richard Hirschman).
It's trivially easy to determine that they're made of amyloid. All you have to do is take specimens and do a Congo Red stain.
SARS-CoV-2 Spike is
known to form fibrinaloid clots resistant to fibrinolysis with considerable ease. It can do this even in platelet-poor blood. These rubber band things are not made up of cells. They're not composed of RBCs and platelets connected by fibrin like a conventional clot. They are hunks of solid protein that have polymerized out of Spike fragments in an autocatalytic biochemical reaction. This is a known property of SARS-CoV-2 Spike. It is a profoundly amyloidogenic protein, to the point where amyloid microclotting is a concern not just in vaccine injury, but in COVID-19 sufferers as well.
Mass spectrometry also showed that when spike protein S1 is added to healthy PPP, it results in structural changes to β and γ fibrin(ogen), complement 3, and prothrombin. These proteins were substantially resistant to trypsinization, in the presence of spike protein S1. Here we suggest that, in part, the presence of spike protein in circulation may contribute to the hypercoagulation in COVID-19 positive patients and may cause substantial impairment of fibrinolysis.
Seven amyloidogenic sequences distributed over the entire S-protein were identified and named according to the starting position in the S-protein (
Figure S2, Supporting Information). All but one (Spike365) of the predicted sequences are in β-sheet conformation in the SARS-CoV-2 Spike cryo-EM structure in its closed state.
(1) The C-terminal part of the protein (Spike1166) is not resolved in the structure.
Solubilized peptides (0.1 mg/mL, PBS pH 7.5, 10% HFIP) were monitored for
in vitro amyloid fibril formation kinetics using ThT, Congo red birefringence (CR), and negative stain transmission electron microscopy (TEM).
Fibrils from most of the synthetic peptides were detected within a few hours by at least one assay (
Table 1,
Figure 1). Spike192, Spike601, and Spike1166 fulfilled all our amyloid criteria: sigmoidal ThT kinetics, Congophilicity, and fibrillar ultrastructure (
Figure 1,
Table 1). Spike192 formed exceptionally well-ordered fibrils comparable to a mix of all peptides (
Figures 1C and
S3C).
There are several short amyloidogenic peptide sequences along the length of SARS-CoV-2 Spike which, when incubated with neutrophil elastase, readily form amyloid fibrils. The digestion of the Spike protein is what kicks off the process. The peptides in question are these little peptide chunks 20 amino acids long, which are tiny compared to the whole protein, which is like 1273 amino acids in length. They are released during the protein's digestion, which can occur as part of an inflammatory response. For instance, neutrophils may attack vaccine-transfected cells via respiratory burst, which would expose those cells and the Spike proteins to neutrophil elastase, a destructive enzyme which busts the Spike apart and releases the amyloidogenic peptides. Other digestive enzymes, like trypsin and cathepsin, may have the same effect.
An enzyme is a protein that snips apart other proteins, like a pair of molecular scissors. It is very common for proteins in the body to begin life as giant, inert molecules that are modified by various enzymes downstream to produce a final product, or a signaling molecule of some type. Immune cells produce tons of damaging radicals and digestive enzymes to break down the structural proteins of viruses and bacteria. Cells have lysosomes that act like recycling centers to bust up unneeded proteins and gather their amino acids into a nutrient pool for loading into tRNAs and translation into new proteins.
Think for a second about what an mRNA vaccine actually does. LNPs loaded with a foreign nucleoside-modified RNA encoding a viral structural protein transfect cells and release their cargo inside, not unlike a virus. These instructions then pass to the ribosomes, which translate them into protein (poorly, thanks to pseudouridylation driving ribosomal frameshifting). The pseudouridylation serves to cloak the modRNA from endosomal toll-like receptors which would otherwise set off an inflammatory cascade to summon immune cells, increasing the toxicity and killing the cells via inflammation before they even have a chance to finish making the product. The process is much akin to how recombinant insulin is made. To make recombinant insulin, they take E. coli or brewer's yeast and they transform them with plasmids for human insulin, culture them in a bioreactor vat, and then collect the protein, purify it, and inject it into diabetics. With the mRNA vaccine, your body is the bioreactor instead, and the "product" is the Spike protein, which the transfected cells display on their surfaces, so that immune cells can recognize the Spike motifs and produce antibodies.
There are many, many problems with this approach. Immune cells do not magically spare human cells that are displaying viral proteins on their surfaces just because they happen to be human. They mercilessly kill the whole cell. This isn't a problem with traditional vaccines, because they either use isolated protein subunits, or weakened virions, and we don't really care about the fate of those.
In conclusion, it is essential to underline that every human cell that intakes the LNPs and translates the viral protein (in case of the mRNA vaccines), or that gets infected by the adenovirus and expresses and translates the viral protein (in case of the adenovirus-based vaccines), is inevitably recognized as a threat by the immune system and killed (Figure
1). There are no exceptions to this mechanism. The severity of the resulting damage and the consequences for health depend on the quantity of the cells involved, on the type of tissue and on the strength of the following autoimmune reaction. For instance, if the mRNA contained in the LNPs would get internalized by cardiac myocytes, and such cells would produce the spike protein, the resulting inflammation would likely lead to the necrosis of the myocardium, with an extent proportional to the number of involved cells. Therefore, it is fundamental to perform pharmacokinetic evaluations in humans, in order to determine the exact biodistribution of the vaccines against COVID-19, and thus to identify the possible tissues at threat.
What do you suppose happens if the LNP vaccine drains through capillaries from the injection site, enters circulation, and winds up in the heart? It's going to transfect heart tissue. This is why people who've taken COVID-19 vaccines are ending up with myocarditis. It's why they're ending up with aortic dissection, from their own immune cells boring a hole through the aorta to kill off cells displaying Spike.
In his mind this catastrophic event is due to the COVID-19 vaccine, and I agree. The mRNA and Spike protein produced by the mRNA circulates in blood on average two weeks, so it is freely able to deposit in the lining of blood vessels and the vascular media of major vessels.
[i ] [ii] Once present, the Spike protein damages cells and incites inflammation which is a destructive process driven by white blood cells, cytokines, and complement. It is known that the second injection is approximately 80-fold more reactogenic with fever, pain, myalgia, etc. As part of that response, there can be a major surge in blood pressure due to release of catecholamines or stress hormones.
[iii] This increase in the change in pressure over the change in time for each heartbeat is the driving force to initiate the tear in the aorta. Once this happens, there is no turning back, the rip goes down the major blood tube and threatens the blood supply to the spinal cord, vital organs, and legs. Each patient is different, with some having external rupture resulting in death. Others require emergency surgery or endovascular stenting to restore blood flow to vital organs. In the case of the optometrist, he was managed conservatively with medications to control blood pressure. Data from the International Registry of Acute Aortic Dissection (IRAD) indicates he faces a 22% 3-year mortality rate and this is increased by his history of prior aortic aneurysm (HR, 2.17; 95% CI, 1.03 to 4.59; P=0.04).
[iv] It is exactly this complication for which I have always advised patients with prior aortic abnormalities (aortopathies) to avoid COVID-19 vaccination. If you know someone who has died shortly after vaccination and they had antecedent back pain or a prior aneurysm, ask the family if there was an autopsy. This is important since aortopathies can be familial and other family members could be screened with imaging and genetic testing. This man’s life is indelibly changed because COVID-19 vaccination was for “keeps.”
Since Spike is amyloidogenic, it's a double-whammy, because now that those cells are dead and spewing their contents everywhere, you have immune cells in the vicinity digesting a protein that readily produces giant protein aggregate clots from its half-digested fragments. Now, you have amyloid nuclei in circulation forming giant rubbery lumps of amyloid and fibrin through autocatalysis.
Amyloid plaques are a feature of Alzheimer's, AL amyloidosis, Lewy body dementia, and so on. They are devastating protein aggregation diseases where proteins that normally have benign functions in the body just start clumping on top of each other without end, forming long strands of inflammatory junk that don't have any purpose whatsoever.
Normally, amyloid fibrils are microscopic. Too small to be seen with the naked eye. A thousand times thinner than a human hair, in fact. What do you suppose it means when amyloid and fibrin form giant arteriovenous casts that are big enough to put in a jar of formaldehyde?
The vaccines aren't even providing any benefits at all, in terms of warding off COVID-19, and that's because of the IgG4 class switch. They're tricking the body into viewing SARS-CoV-2 Spike as a harmless allergen and ignoring it.
To borrow, and extend, a brilliant analogy from
Igor Chudov, who said it best: consider a home invasion. The virus is a burglar, and that burglar is coming to take your stuff. Thus far, nobody has been home, but the alarms have still been working; the original antigenic sin caused by outdated mRNA has thus far precluded a fully effective antibody response, but the alarm is still on, and the cops are going to show up eventually. Their diseases have been more severe and lasted longer, but they've still been, for the vast majority, generally able to survive. That's about to change.
The situation we now find ourselves in is much worse. Not only is nobody home anymore, but the cops are standing by the door,
guarding the burglars. IgG4 antibodies act to suppress the innate immune system as well as the adaptive: they bind to FcγRIIB complexes on cell surfaces that variously deactivate and destroy immune responses and cells, they bind to the virions themselves, they bind to CD4 T-cells and dendritic cells, and and they even bind to the signal cascade transducers of the complement immune system; everywhere they go, the message is loud and clear.
Nothing to see here. Please disperse.
I had hoped that at least elements of the innate immune system would be spared. No dice. With even
complement being shut down, vaccinees are in for a world of hurt. Consider the excess death statistics that have been steadily rising lately: while these are, thus far, very likely to have been attributable mostly to otherwise silent vaccine injuries, the next time the COVID numbers start rising, we should expect to see vaccinees dying in droves. They likely, mostly, won’t be dying in hospital, either: with the immune response so dramatically blunted, the typical signs and symptoms of infection will likely be all but absent. It would actually be funny, if it weren’t so horrific: uncontrolled replication will lead to trillions of virions tearing unchallenged through the tissues, exposing the victim to a similar quantity of bioactive, toxic spike protein as is created by the mRNA shots. Blood clots, myocarditis, autoimmunity, prion diseases; everything that the shots can cause in the short term, so too will the virus; and
the very first clue that they’re even infected may well be their sudden and unceremonious death, perhaps preceded by some general feeling of being unwell if they’re lucky.
RNA vaccines are efficient preventive measures to combat the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) pandemic. High levels of neutralizing SARS-CoV-2 antibodies are an important component of vaccine-induced immunity. Shortly after the initial two mRNA vaccine doses, the immunoglobulin G (IgG) response mainly consists of the proinflammatory subclasses IgG1 and IgG3. Here, we report that several months after the second vaccination, SARS-CoV-2–specific antibodies were increasingly composed of noninflammatory IgG4, which were further boosted by a third mRNA vaccination and/or SARS-CoV-2 variant breakthrough infections. IgG4 antibodies among all spike-specific IgG antibodies rose, on average, from 0.04% shortly after the second vaccination to 19.27% late after the third vaccination. This induction of IgG4 antibodies was not observed after homologous or heterologous SARS-CoV-2 vaccination with adenoviral vectors. Single-cell sequencing and flow cytometry revealed substantial frequencies of IgG4-switched B cells within the spike-binding memory B cell population [median of 14.4%; interquartile range (IQR) of 6.7 to 18.1%] compared with the overall memory B cell repertoire (median of 1.3%; IQR of 0.9 to 2.2%) after three immunizations. This class switch was associated with a reduced capacity of the spike-specific antibodies to mediate antibody-dependent cellular phagocytosis and complement deposition. Because Fc-mediated effector functions are critical for antiviral immunity, these findings may have consequences for the choice and timing of vaccination regimens using mRNA vaccines, including future booster immunizations against SARS-CoV-2.
Keep in mind, Denis Rancourt's estimates claim that the shots are killing 1 in 800 people for every dose:
That's tens of millions dead, so far. Covered up by statistical tomfoolery carried out by complicit authorities with a clear depopulation agenda.
This is a shitshow of utterly biblical proportions. Nothing else in human history even comes close. To be pro-COVID-19 vaccine is to engage in apologia for the small army of VHEMT-tier, nihilistic, mass-murdering motherfuckers who have taken over our institutions and are puppeteering them from the inside out.
