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It's the childrens fault! I only did what they wanted me to do!
Megathread SRS and GRS surgeons and associated horrors - the medical community of experimental surgeons, the secret community of home butchers
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Titanium is wacky, it's not used because it's inert, it's actually used because your body treats it like calcium and you get bone deposition/remodelling at the surface of the metal in an interesting oxidation. Very useful for implants. It's a fascinating little quirk.
I only know one FtM. The amount of medical care she sucks up like a leech is worse than most of the munchies. She is entirely useless. Can't hold down any kind of job, misanthropic, just... Ugh. It's such a waste.
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How would you even insert the erection pump into the clitballs?? Because the mutated clit sits there. There has to be something going wrong at some point. I mean what the mad chink butcher created is already wrong and should never exist but there is only so much this crotch abomination can take until it' falls apart after another sensless surgery.
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So, it seems like she developed arterial thrombosis, which according to this book means "A new recipient vessel must be found, eventually requiring an arterial interposition"
Either way, pics from the book, showing salvages:
Fig. 13-1 The DIEA (deep inferior epigastric artery) and venae comitantes (green backgrounds) are isolated through the prolonged groin incision. A microvascular clamp is placed on the vessels before division.
Fig. 13-2 The deep inferior epigastric artery and vein are tunneled into the defect after an extended Pfannenstiel incision is used to harvest the vessels. Two saphenous veins as outflow vessels, the left ilioinguinal nerve, and the denuded clitoris are also shown.
Fig. 13-3 The transposed DIEA and venae comitantes almost reach the recipient site. It is not always necessary to take such a long stump. The shorter the stump, the bigger the caliber.
Fig. 13-4 The left descending branch of the lateral circumflex femoral artery is dissected and tunneled in the defect for recipient vessels when inferior epigastric arteries were not useful and femoral arteries had extensive scarring.
Fig. 13-5 A, A ventral view of an ALT plus superficial circumflex iliac artery perforator (SCIAP) phalloplasty. The skin graft (covered with SurfaSoft) was placed on the ventral side (thus on the SCIAP) to avoid tension while closing.
B, One year after surgery, the skin graft on the ventral side is not always visible and has an acceptable appearance.
Fig. 13-6 A, When an arteriovenous loop is created, the phallus looks congested. This is normal.
B, After closure of the fistula, the skin color returns to normal.
Fig. 13-7 A, A rectus sheath hematoma after inferior epigastric flap harvest for RFF phalloplasty.
B, A CT scan confirmed the findings. The patient was taken to the operating room on an emergency basis to avoid vascular compromise of the flap.
Fig. 13-8 A pedicled ALT flap for a phalloplasty converted to free flap on day 1 because of vascular insufficiency, resulting from tension on the pedicle. The pedicle is divided and anastomosed to the groin vessels (femoral artery and great saphenous vein; see text for details).
Fig. 13-9 A, Partial necrosis in an RFF phalloplasty. After escharotomy the healthy dorsal flap is used (like a dorsal preputial flap is used in hypospadias repair) to reconstruct a glans and a meatus.
B, The ventral side can be skin grafted.
Fig. 13-10 A, After partial failure of urethral reconstruction, the missing half can be reconstructed with a skin graft.
B, A sterile sleeve, such as the one used for gamma probes, is filled with gel and inserted to hold the graft in place.
Fig. 13-11 A, Frontal view of an RFF used to reconstruct the whole urethra.
B, An external skin island is left after tubing to reconstruct the ventral side.
Fig. 13-12 A, This patient developed a penoscrotal fistula and infection 3 weeks after RFF phalloplasty and was treated with debridement and an immediate gracilis flap to the defect within 1 week.
B, The patient urinated well 3 months after phalloplasty.
The book this comes from, "Gender Affirmation: Medical & Surgical Perspectives", has some of our thread regulars like Crane, Bowers and Miroslav as contributors.
Bonus: This sad shriveled "cock"
Another possibility, which seems is the one from this article is distal arteriovenous fistula
Either way, it's an interesting procedure so here are the pics:
Figure 1. A, B, Recruiting the deep median cubital vein (4) as part of the vascular pedicle of the radial forearm flap, will insure drainage of both the comitant veins of the radial artery (3) and the superficial cephalic vein (2) through one venous anastomosis of the median cubital (1), or even brachial, vein.
Figure 2. In the inguinal-pubic receptor area, an arteriovenous loop was created by end-to-side anastomosis of the distal end of a 22-cm length of the left greater saphenous vein to the femoral artery at the level of the femoral triangle.
Figure 3. The fistula between the distal arterial end and a superficial branch of the cephalic vein in the neophallus was readily available to Doppler-ultrasound inspection postoperatively. The lighter line indicates the radial artery; the darker one, the cutaneous vein. Recruiting a subcutaneous vein further allowed for "taking the pulse" of the neophallus manually
Figure 4. In order to prevent unnecessary postoperative morbidity and scarring, we routinely harvest the length of saphenous vein through three or four small transverse incisions.
Oh, book also has this passage on puberty blockers:
"GnRH agonist medications have been used extensively in this age group for the treatment of precocious puberty for more than 25 years and are considered safe and reversible. In the transgender population, theoretical risks include reduced bone mineral density accrual while the patient is receiving treatment (this has been found to improve after treatment with cross-sex hormones) and the unknown impact on brain maturation while the patient is on suppression. The concern about brain maturation may be overstated, given that this is not a significant concern for children with constitutional delay of puberty."
Their sequence of appropriate treatment as the child ages:
Fig. 14-5 General sequence for medical and surgical management of transgender adolescents. The mental health provider supports the child and family, treats comorbid mental health conditions, and helps with the logistics of social transition throughout adolescence.
I'm looking back and not only do they NOT GIVE A GOOD ARGUMENT for even starting treatment, feels like they do the opposite:
Children are born into a gendered world, where boys and girls are often dressed differently and encouraged to participate in different types of play, and where men and women may traditionally assume different familial or occupational roles. In addition, stereotypical gender roles and gender behavior vary among different cultures and also change over time. Children 2 to 3 years of age are able to label themselves as boy or girl; by ages 4 to 5, they can understand the stability and lasting nature of gender. Gender-specific oy preference emerges as young as 12 months, and children can use gender labels (boy, girl) by 2 years of age. As young children begin to develop preferences in play and dress, these behaviors may appear very gender conforming, very gender nonconforming, or somewhere within this spectrum. Some gender nonconforming children may state a desire to be the other gender or express a feeling that they are the other gender, even by 3 years of age. Young children with cross-gender identifi ation may become transgender adolescents; however, the evidence suggests that a larger percentage will not be transgender, and that individuals with a cross-gender identity in young childhood that later desists have high rates of identification as gay or lesbian.
Sex hormones, chiefly testosterone and estrogen, are steroids produced by the testes and ovaries that cause a multitude of effects that result in biologic differences between males and females. In fetal life and also in the fi st 6 to 12 months of postnatal life, there are signifi ant differences in sex hormone levels in male and female fetuses and infants. The absence of testosterone production in fetal life results in normal female genitalia, whereas testosterone produced in the fetal testes is converted to dihydrotestosterone in the genital tissue, resulting in virilization of the external tissues into normal male genitalia. Differences in sex hormone levels during fetal life and infancy between the biologic sexes also likely play an important role in sexual differentiation in brain organization. These differences may be an important contributor to group differences in behaviors observed between males and females later in life.
In later childhood, as gender identity begins to manifest, the testes or ovaries have entered a quiescent stage with very little sex hormone production, and therefore there is little difference in the hormonal milieu between prepubertal male and female children (Figs. 14-1 through 14-3). Therefore hormonal intervention is not indicated for prepubertal children. Instead, the child and family can focus on mental health and logistical issues, such as addressing mental health comorbidities (for example, anxiety or depression) and deciding whether to make a social transition to the affirmed gender in young childhood. Although there is consensus that prepubertal children with gender dysphoria should be seen by a mental health professional with gender experience, there is not a consensus among mental health providers with respect to the goals of treatment.15 Some argue that because of the frequency of desistance later in adolescents, the therapeutic goals should focus on reduction in dysphoria through acceptance of the biologic sex.Another strategy focuses less on gender identity but rather on emotional, behavioral, and family problems that are co-occurring. Finally, affirmative approaches help families to support the child’s identifi d gender and assist children and families in making a social transition.18 When prepubertal children make a social transition, presenting themselves as their affirmed gender, their ability to “pass” as their affirmed gender is aided by the fact that they have not yet developed secondary sexual characteristics. Th s process of “passing” is also known as gender attribution, the process an observer undertakes when deciding what gender they believe another person is.
Sex hormones, chiefly testosterone and estrogen, are steroids produced by the testes and ovaries that cause a multitude of effects that result in biologic differences between males and females. In fetal life and also in the fi st 6 to 12 months of postnatal life, there are signifi ant differences in sex hormone levels in male and female fetuses and infants. The absence of testosterone production in fetal life results in normal female genitalia, whereas testosterone produced in the fetal testes is converted to dihydrotestosterone in the genital tissue, resulting in virilization of the external tissues into normal male genitalia. Differences in sex hormone levels during fetal life and infancy between the biologic sexes also likely play an important role in sexual differentiation in brain organization. These differences may be an important contributor to group differences in behaviors observed between males and females later in life.
In later childhood, as gender identity begins to manifest, the testes or ovaries have entered a quiescent stage with very little sex hormone production, and therefore there is little difference in the hormonal milieu between prepubertal male and female children (Figs. 14-1 through 14-3). Therefore hormonal intervention is not indicated for prepubertal children. Instead, the child and family can focus on mental health and logistical issues, such as addressing mental health comorbidities (for example, anxiety or depression) and deciding whether to make a social transition to the affirmed gender in young childhood. Although there is consensus that prepubertal children with gender dysphoria should be seen by a mental health professional with gender experience, there is not a consensus among mental health providers with respect to the goals of treatment.15 Some argue that because of the frequency of desistance later in adolescents, the therapeutic goals should focus on reduction in dysphoria through acceptance of the biologic sex.Another strategy focuses less on gender identity but rather on emotional, behavioral, and family problems that are co-occurring. Finally, affirmative approaches help families to support the child’s identifi d gender and assist children and families in making a social transition.18 When prepubertal children make a social transition, presenting themselves as their affirmed gender, their ability to “pass” as their affirmed gender is aided by the fact that they have not yet developed secondary sexual characteristics. Th s process of “passing” is also known as gender attribution, the process an observer undertakes when deciding what gender they believe another person is.
Nothing in this text makes me believe puberty blockers are a good idea, and THE ARTICLE THEY USE TO SAY IT'S SAFE is talking about VERY young children developing puberty before they should (Girls younger than 8, boys younger than 10), one example is a 4 year old girl for Christ's sake, they're advoctaing for children as old as 14 years old on those blockers! That's not what their own "proof" is about! The other article about it is a study of the first 21 child's development in only 2 years, nothing after they're adults and again ONLY 21, c'mon.
This all proves almost nothing, no wonder Bowers is already changing his mind, shame the people that aren't reading their own "sources" will never learn
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Lmao, that last twisted black “dick” with the pee coming out at an angle from the top is just hilarious and sad.
Crepidodera fulvicornis
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Oh my fucking god, this is a great picture for “kill it with fire” meme.
Art Kassul
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"I revised my glans"
We may make our glans, but God has the last word.
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The most effective defense against a hadouken.
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In a single thread about bad SRS results.
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- Jan 26, 2020
The trannies can seethe all they want. It's their content, their pictures, their 'lived experiences' that they're so goddamn fond of.
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Cope, seethe, dilate, you'll never be a woman and all that jazz fags.
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- Apr 23, 2021
Some bonus on hormones: a study on gonadroprin to treat adolescent endometriosis (so correct age group):
Long-Term Effects of Gonadotropin-Releasing Hormone Agonists and Add-Back in Adolescent Endometriosis
"Results: The response rate was 61%. Almost all (96%) reported side effects during treatment; 80% reported side effects lasting > 6 months after stopping treatment. Almost half (45%) reported side effects they considered irreversible, including memory loss, insomnia, and hot flashes. Despite side effects, subjects rated GnRHa plus add-back as the most effective hormonal medication for treating endometriosis pain; two thirds (16/25) would recommend it to others. More subjects who received a modified two drug add-back regimen versus standard one drug add-back would recommend GnRHa and felt it was the most effective hormonal medication."
Two thirds would reccomend to others? There's a caveat:
"Willingness to recommend GnRHa therapy to others was a more common response among subjects who received a modified add-back regimen of norethindrone acetate plus low dose estrogens rather than the more common add-back regimen of norethindrone acetate alone"
So, girls that were also receiving estrogen had better results, which is not the case for trans children. Further on that:
"These results provide further support for the previously published outcomes from this patient cohort; a two-drug add-back regimen was found to be superior for preserving bone mineral density and preventing menopausal side effects during treatment."
Percentage of adolescents with endometriosis who would recommend GnRHa + addback therapy to a friend or family member who had painful endometriosis, by add-back group
Of course, it goes too that it's a small group, but the point is that it's still not clear if it's safe or not, reports are mixed
Long-Term Effects of Gonadotropin-Releasing Hormone Agonists and Add-Back in Adolescent Endometriosis
"Results: The response rate was 61%. Almost all (96%) reported side effects during treatment; 80% reported side effects lasting > 6 months after stopping treatment. Almost half (45%) reported side effects they considered irreversible, including memory loss, insomnia, and hot flashes. Despite side effects, subjects rated GnRHa plus add-back as the most effective hormonal medication for treating endometriosis pain; two thirds (16/25) would recommend it to others. More subjects who received a modified two drug add-back regimen versus standard one drug add-back would recommend GnRHa and felt it was the most effective hormonal medication."
Two thirds would reccomend to others? There's a caveat:
"Willingness to recommend GnRHa therapy to others was a more common response among subjects who received a modified add-back regimen of norethindrone acetate plus low dose estrogens rather than the more common add-back regimen of norethindrone acetate alone"
So, girls that were also receiving estrogen had better results, which is not the case for trans children. Further on that:
"These results provide further support for the previously published outcomes from this patient cohort; a two-drug add-back regimen was found to be superior for preserving bone mineral density and preventing menopausal side effects during treatment."
Percentage of adolescents with endometriosis who would recommend GnRHa + addback therapy to a friend or family member who had painful endometriosis, by add-back group
Of course, it goes too that it's a small group, but the point is that it's still not clear if it's safe or not, reports are mixed
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- Jan 31, 2015
Good. Let people read what's going on with trans genital multilation.
This man is so fucking r.etarded. He is drawing attention to this thread which means more people are going to read it and that might convince a few to NOT run to the genital butcher.
Well done, tard faggot.
![LinkinParkxNaruto[AMV]](http://uploads.kiwifarms.st/data/avatars/l/61/61177.jpg?1699571951){kind=avatar}
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Legit questions. Does "good" results even exist? Everything you see on their own subs is on the scale from bad to worse.
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- Sep 11, 2021
That dick gonna fall off in public and give a veteran a heart attack.
PresidentTerf
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Ah the absolute nutters from gender cynical, that takes me back, still pathological as ever I see.
Would have thought they'd be too busy with TiA going full Terf.
I agree with the above user, where are the actual good results?
That person with every sexual organ possible on her crotch seems to be the closest candidate.
Outwith Quiddany
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