@mdrop22 Washington state ferry workers, you say...
@Drain Todger this is truly the lolcow arc we need right now.
@mrdrop22
@Fanatical Pragmatist
Something like 100+ engine crew are opposed to Inslee's mandate.
Ship crews are not easy to replace. Number of reasons why.
First of all, the cabotage laws codified by the Jones Act say that US-flagged vessels must have American crews, period. They can’t bring in guys on work visas as strikebreakers.
Second, we’re unionized contractors. Closed shop. You have to be a member of a union. So, their choice of crews is limited by what our unions can supply.
Third, the credentialing process put in place by the US Coast Guard for US Merchant Mariners requires mandatory on-the-job experience for set lengths of time in order to obtain each license, known as “sea time”. In order to become a QMED, you need 180 days of training spent on an actual boat (usually from a maritime academy of some sort), followed by a multiple-choice exam with a minimum 70% correct answers to pass (literally everyone passes this test by
using license prep software and doing practice exams until they pass consistently, so you can be basically the most retarded mouth-breathing grug on the planet and pass), and a physical. This is mandatory. No exceptions. You cannot just hire some random guy off the street, because first, he has to have 180 days minimum of training just to be an Oiler. Then, he has to join a union. Then, he needs basic safety training and basic firefighting. And all of this is what you need to be an entry-level QMED-Oiler.
Now, most guys who join one of the unions (there are different ones for engineers, deck crew, captains/mates, et cetera) will “ship out”. They’ll take a job out of the union hall for a big shipping company, tug and barge, oil and gas, whatever, and they’ll sail for six months solid, way out at sea. They live aboard ship. When their watch is over, they go to their bunk in their quarters and rack out. WSF is different. Being an inland waters job, unless you live all the way off in bumfuck Oregon and sleep on the boat and then drive back down to Portland or Medford or whatever every week, you’re never really that far from home. What this means is that rather than living aboard the ship, you get to go home every night. To be honest, the pay ain’t too hot, as far as oiling goes. Deep sea, you can make a little bit more money. However, a lot of guys like the idea of sleeping in their own bedroom every night, having a brewski, playing some fucking Xbox, and passing out, whatever. Can’t really do that when you’re deep sea. Your only entertainment out there is old, yellowed Clive Cussler novels and a quick wank in the shower.
Now, if someone hires on as an Oiler with WSF, the prospective job candidate must be insane enough to want to work “On-Call” for a year or three before getting a permanent position. At WSF, there are usually one or two dozen on-call oilers whose only job is to fill in for guys who call out sick. These poor bastards have absolutely no work-life balance, because they have no idea when they have to work and can’t make plans for shit. Dispatch’s job is to give them seven 12-hour days in a 14-day period, on basically any vessel in the fleet. These can be either day or night shift, in any order imaginable, so long as there is a one-day gap for sleep between switching from a day to night shift. In other words, it is quite possible for an on-call oiler to be called for three days, get a day off, two nights, another day off, and then two more days, and the rest of the 14-day period off, all on entirely different boats with relieving ports that could be as little as a ten-minute drive from one’s house, or as far as a couple hours away. If people don't call out sick, on-calls don't get work. It is entirely possible to have only two days of work in a 14-day period, get a short paycheck, find your fridge is empty and so is your bank account, and then eat your fucking parakeet while shivering in the corner of your freezing hovel.
When on-calls go permanent and get assigned to a vessel, they work my schedule, which is basically a DuPont Shift Schedule; four shifts, A, B, C, D. Week one, A and B relieve each other while C and D are off. The next week, C and D relieve each other while A and B get the week off. The on week is 7 consecutive 12-hour days, followed by a week off, then 7 consecutive 12-hour nights, followed by a week off, rinse and repeat. This comes out to an 84-hour week of time actually on the clock, but in practice, if you include the commute, it is a 100+ hour week, easily. Especially if you have to deadhead, which is when you commute to the start of your watch by riding the boat unpaid, off-the-clock (airline attendants know this pain all too well). For me, my door-to-door time is usually something like fifteen hours, but if we're running really late, another hour or so gets tacked onto that. On days, we mostly do rounds of the engine room to make sure everything is running right and isn't leaking. On nights, we perform maintenance and stop and start the main engines. WSF vessels are double-end RO-ROs that take cars and passengers. There is a pilothouse, rudder, and prop at each end (yes, you can oppose the rudders and crab sideways, or practically rotate in place, it's really cool), and when we leave a terminal, we don't turn around. The bridge crew just walks to the other pilothouse and transfers the control to that one, and then the stern becomes the bow, and off we go. These boats are generally quite reliable. They don't run themselves, but major engineering casualties are uncommon (unless an outside contractor ratfucked something during a major overhaul, or the warehouse gave us defective spare parts, both of which I've seen the consequences of; a lot of spare parts for these older diesels just aren't the same quality they used to be). As you can probably imagine, the first few days of the off week are generally spent catching up on lost sleep.
Calling out sick at WSF is not the same as calling out sick at other jobs. You accrue paid sick leave hours in addition to vacation and comp time, and when you call out sick, those hours go on your pay order, for the same length as an actual shift; 12 hours. You can call out sick for basically anything. You can call out sick because you had insomnia, or because your knee was hurting, or because you ate day-old pasta and had a stomach ache, or because you just didn't feel like coming to work that day. Dispatch does not care. An on-call will fill in for you. However, if lots of guys call out sick at once, and the pool of on-call workers is depleted, they start calling the permanent guys who are on our off weeks and begging us to do overtime, which we have the contractual right to refuse. In fact, I can literally let them go to voicemail with no penalty whatsoever, put my ringer on silent, roll over, go back to sleep, and wake up feeling refreshed and happy that I didn’t have to go spend twelve hours of my life on a fucking boat. If enough people were to call out sick at once, they would quite literally have no choice but to tie up the boats for lack of crew. You cannot sail a vessel that is not fully manned. The Coast Guard won't let you. The Coast Guard COI sets the minimum manning requirements. If there is one man less than those requirements, you cannot sail, period. All it can take to tie up a boat is one guy being a no-show. The guy currently on watch whose relief has not shown starts accruing overtime for a few hours. After three hours, if Dispatch cannot find a relief, the boat is tied up and he goes home.
A QMED-Oiler is a “Qualified Member of the Engineering Department”, as in, an unlicensed marine engineer rating, kind of like being enlisted versus being an officer in the military. We have a different union contract from the licensed engineering officers; the Assistant Engineers and Chief Engineers. Licensed engineers need a great deal of sea time just to become licensed. You need 1080 days of sea time, with 720 days spent as QMED or equivalent, followed by an extensive written test and an advanced firefighting course, just to become a 3rd Assistant Engineer. Then, when you get the license, you are not promoted instantly. Instead, you go on a promotion list based on seniority, where you have to wait another few years to actually get fucking promoted. Ideally, you accrue sea time on a large vessel with some beefy engines, because guess what? There’s a horsepower limitation on some engineer licenses. If you end up with a limitation, you can’t work vessels with horsepower any higher than that. The requirements for Chiefs are even more stringent than that, and require some time spent as an Assistant. There is no requirement to promote, so if someone wants to stay an Oiler or Assistant forever, they can do that. In order to advance, someone has to actually want to advance and get the license, and if they're a fresh Oiler, it's basically 6 years from the hire date for them to become a permanently assigned Assistant Engineer, bare minimum. So you see, if a bunch of officers quit or are fired all at once, they're
fucked.
So, in order to work at WSF, you have to have American citizenship, you have to have a USCG credential that takes ages to acquire, you have to be union, and you have to be insane enough to want to work hundred-hour weeks on a regular basis.
With all this in mind, I want you to imagine the thermonuclear shitstorm that would ensue if Inslee actually fired a full third of us for refusing the vax.
Where will he find a hundred schizoid mechanics on short notice? Or replacements for the thousands of cops, firefighters, teachers, and other state employees who are standing our ground and refusing to vaccinate?
Inslee will fucking blink. He has no choice.
I guarantee you a good portion of these retards calling Ivermectin "horse medication" have snorted some bumps of ketamine at the last concert they went to. I personally know people like this.
We must take the pony paste. It’s the only way.
Seriously, though. Doesn’t it seem like this whole narrative of Ivermectin as a veterinary antiparasitic drug is, like,
really fucking artificial and forced? It comes in pill form for humans, for fuck’s sake. It’s not ”horse dewormer”. If it stops SARS-CoV-2, then clearly, the mode of action is separate from its antiparasitic effect.
The media would have you believe that drugs can only have very specific, clearly defined effects. This is plainly wrong. Hell, even scientists make the same mistake. I have seen papers where they’ve tried H2 blockers like famotidine for COVID-19, and then speculated that the virus has a histamine-promoting effect that causes inflammation. Actually, famotidine, cimetidine, and ranitidine are all antioxidants and all scavenge reactive oxygen species. And so does Melatonin. And Indomethacin, too, which acts as a free iron chelator for fuck’s sake.
One substance, multiple effects. Just because Ivermectin is an antiparasitic, that doesn’t mean it doesn’t have an antiviral mechanism, too.
Ivermectin for COVID-19: real-time analysis of all 101 studies
c19ivermectin.com
What’s that? It only works if you give it to people before they get sick? Gee, I wonder why?
Oh wait. The viral load peaks right when you become symptomatic? And all these clinical trials where they test Ivermectin on severely ill people in hospitals with hyperinflammation had no benefit, because the viral load had tapered off already and it was all just the immune system attacking damaged tissues? Wait a minute, what the hell are these clinical trialists doing?
Let’s see, this is the review the NYT are citing. What does it say?
Okay, so it hypothetically blocks importin and prevents nuclear import, which means it has antiviral activity. What else? What are the conclusions of the study?
We’re uncertain
We’re uncertain
We’re uncertain
Meanwhile, all the placebo-controlled randomized clinical trials of Ivermectin (and basically all other antivirals, too) are literally of people who are fucking dying of hyperinflammation and sepsis akin to an acute autoimmune attack, who have already cleared most of the virus from their systems and are now experiencing delayed inflammatory response.
Let me repeat; the intervention is too fucking late. This is like shocking flatline with a defibrillator, watching the patient sit there with their tongue hanging out and Xes over their eyes, and then claiming that defibrillators don’t work and should not be used for V-fib. It’s vapid and ludicrous. There are no trials of antivirals as prophylaxis against the virus, nor have they examined prophylactic antiviral users and observed their response to the drug when and if they develop illness. These RCTs literally all consist of people who are already quadraspazzed on a life-glug. The antiviral therapy is being started when the virus is already fucking gone.
Too. Fucking. Late. For. Antivirals.
The only treatments that work on people in that condition are immunomodulatory. Steroids, antioxidants, monoclonal antibodies that inhibit cytokines, et cetera. It’s too fucking late for antivirals. That’s why Remdesivir doesn’t work, nor Kaletra, nor HCQ, nor Ivermectin. It’s too fucking late! My god, are these people braindead? Look at the PCR. Look at the viral titers. When the inflammation sets in, the virus is almost all gone. Gone!
What are they trying to prevent the replication of? Ludicrous! There is a tiny window to take antivirals and have a benefit, and that’s
immediately post-exposure, during the entire incubation period
. See for yourself. Look up COVID-19 clinical course, and look at the fucking figures. Every one of those papers has the same exact figure. Viral response starts off high, tapers off. Then, host inflammatory response comes on as a delayed effect.
People aren’t seeking treatment until it’s too late to do anything about it. They become symptomatic, they wait five days and tough it out thinking it’s just a flu bug, and in most cases, that’s what it is. It resolves, they get over it, they get robust immunity, and it’s no big deal. On the other hand, a small minority of people with pre-existing endothelial dysfunction develop hyperinflammation and sepsis, turn blue in the face, and then go to the ER, by which point antivirals are not going to do a fucking thing. This small minority of people developing hyperinflammation are pretty much the entirety of the goddamn cohort in these randomized clinical trials for antivirals. As in, this is
complete fucking bullshit.
And yet, the media keeps reading the conclusions of these bullshit studies, and they’re like “See? Lol, antivirals don’t work! Just get the jab!”
How are they giving people Ivermectin in India?
Background: Ivermectin is one among several potential drugs explored for its therapeutic and preventive role in SARS-CoV-2 infection. The study was aimed to explore the association between ivermectin prophylaxis and the development of SARS-CoV-2 infection among healthcare workers.
Methods: A hospital-based matched case-control study was conducted among healthcare workers of AIIMS Bhubaneswar, India, from September to October 2020. Profession, gender, age and date of diagnosis were matched for 186 case-control pairs. Cases and controls were healthcare workers who tested positive and negative, respectively, for COVID-19 by RT-PCR. Exposure was defined as the intake of ivermectin and/or hydroxychloroquine and/or vitamin-C and/or other prophylaxis for COVID-19. Data collection and entry was done in Epicollect5, and analysis was performed using STATA version 13. Conditional logistic regression models were used to describe the associated factors for SARS-CoV-2 infection.
Results: Ivermectin prophylaxis was taken by 76 controls and 41 cases. Two-dose ivermectin prophylaxis (AOR 0.27, 95% CI, 0.15-0.51) was associated with a 73% reduction of SARS-CoV-2 infection among healthcare workers for the following month. Those involved in physical activity (AOR 3.06 95% CI, 1.18-7.93) for more than an hour/day were more likely to contract SARS-CoV-2 infection. Type of household, COVID duty, single-dose ivermectin prophylaxis, vitamin-C prophylaxis and hydroxychloroquine prophylaxis were not associated with SARS-CoV-2 infection.
Conclusion: Two-dose ivermectin prophylaxis at a dose of 300 μg/kg with a gap of 72 hours was associated with a 73% reduction of SARS-CoV-2 infection among healthcare workers for the following month. Chemoprophylaxis has relevance in the containment of pandemic.
Oh wow. What a surprise. They’re giving it to people before they even get sick. Pre-emptively. As in, prophylaxis. I’m shocked.
Shocked, I tell you.
In that letter I sent to the CDC and HHS in February of 2020, I tried warning them about the possibility of ADE. To quote myself, from my letter dated Feb. 25th, 2020:
SARS-CoV can, in vitro, cause antibody-dependent enhancement, where a weak antibody response similar to the kind found in Dengue carries the virus to the Fc receptor pathway and allows it to infect immune cells. Your own immune cells aid viral replication instead of preventing it like they’re supposed to.
I drew that conclusion from one of Shi "The Bat Lady" Zhengli's own papers.
Antibody-dependent enhancement (ADE) of viral entry has been a major concern for epidemiology, vaccine development, and antibody-based drug therapy. However, the molecular mechanism behind ADE is still elusive. Coronavirus spike protein mediates viral entry into cells by first binding to a receptor on the host cell surface and then fusing viral and host membranes. In this study, we investigated how a neutralizing monoclonal antibody (MAb), which targets the receptor-binding domain (RBD) of Middle East respiratory syndrome (MERS) coronavirus spike, mediates viral entry using pseudovirus entry and biochemical assays. Our results showed that MAb binds to the virus surface spike, allowing it to undergo conformational changes and become prone to proteolytic activation. Meanwhile, MAb binds to cell surface IgG Fc receptor, guiding viral entry through canonical viral-receptor-dependent pathways. Our data suggest that the antibody/Fc-receptor complex functionally mimics viral receptor in mediating viral entry. Moreover, we characterized MAb dosages in viral-receptor-dependent, Fc-receptor-dependent, and both-receptors-dependent viral entry pathways, delineating guidelines on MAb usages in treating viral infections. Our study reveals a novel molecular mechanism for antibody-enhanced viral entry and can guide future vaccination and antiviral strategies.
And now, there's this:
COVID-19 (SARS-CoV-2) disease severity and stages varies from asymptomatic, mild flu-like symptoms, moderate, severe, critical, and chronic disease. COVID-19 disease progression include lymphopenia, elevated proinflammatory cytokines and chemokines, accumulation of macrophages and neutrophils in lungs, immune dysregulation, cytokine storms, acute respiratory distress syndrome (ARDS), etc. Development of vaccines to severe acute respiratory syndrome (SARS), Middle East Respiratory Syndrome coronavirus (MERS-CoV), and other coronavirus has been difficult to create due to vaccine induced enhanced disease responses in animal models. Multiple betacoronaviruses including SARS-CoV-2 and SARS-CoV-1 expand cellular tropism by infecting some phagocytic cells (immature macrophages and dendritic cells) via antibody bound Fc receptor uptake of virus. Antibody-dependent enhancement (ADE) may be involved in the clinical observation of increased severity of symptoms associated with early high levels of SARS-CoV-2 antibodies in patients. Infants with multisystem inflammatory syndrome in children (MIS-C) associated with COVID-19 may also have ADE caused by maternally acquired SARS-CoV-2 antibodies bound to mast cells. ADE risks associated with SARS-CoV-2 has implications for COVID-19 and MIS-C treatments, B-cell vaccines, SARS-CoV-2 antibody therapy, and convalescent plasma therapy for patients. SARS-CoV-2 antibodies bound to mast cells may be involved in MIS-C and multisystem inflammatory syndrome in adults (MIS-A) following initial COVID-19 infection. SARS-CoV-2 antibodies bound to Fc receptors on macrophages and mast cells may represent two different mechanisms for ADE in patients. These two different ADE risks have possible implications for SARS-CoV-2 B-cell vaccines for subsets of populations based on age, cross-reactive antibodies, variabilities in antibody levels over time, and pregnancy. These models place increased emphasis on the importance of developing safe SARS-CoV-2 T cell vaccines that are not dependent upon antibodies.
Oh shit, here we go:
New evidence, including sworn affidavits from leading experts such as Professor Luc A. Montagnier, has been submitted to the International Criminal Court by law
dailyexpose.co.uk
A new claim has also been submitted to the ICC due to the vast amount of new evidence and information that has come to light in the past few months, and the lawyers say they now have compelling evidence that “the SARS-CoV-2 virus and the Covid-19 ‘vaccines’ are deliberately engineered bioweapons that have been released in two phases on unsuspecting peoples of the world”.
Attorney Melinda C. Mayne, and Kaira S. McCallum have also confirmed that they have now be joined by lawyers who have filed similar Requests for Investigation to the International Criminal Court, in France, the Czech Republic and Slovakia.
Never forget that the vaccine and the virus were made by the same people, and they did it all with American taxpayer dollars.
British scientist Peter Daszak's organisation channelled cash to Wuhan scientists to do the Gain-of-Threat experiments that caused the Covid-19 Pandemic.
peterdaszak.com
U.S. Gov't Sent "mRNA Coronavirus Vaccine Candidates" to University Researchers WEEKS BEFORE "COVID" Outbreak in China! A confidentiality agreement shows pot
dailyexpose.co.uk
Look at the timeline. There was a moratorium on federal funding for SARS gain-of-function research in 2014. The research continued, in defiance of the moratorium. Anthony Fauci, the NIH/NIAID, DTRA, and USAID funneled millions of dollars to the Wuhan Institute of Virology and Shi Zhengli, laundering the grant money through Peter Daszak's EcoHealth Alliance. Ralph Baric and Shi Zhengli were colleagues and co-wrote papers together. Ralph Baric mentored Shi Zhengli in the art of modifying viruses by serial passage, leaving no trace. When the virus sickened workers at the WIV in November of 2019, Peter Daszak knew immediately what had happened. A month later, in December 2019, before a pandemic had even been announced, Ralph Baric took delivery of confidential coronavirus mRNA vaccine materials co-owned by Moderna and NIH. A month after that, in January of 2020, not only did China finally announce the outbreak, they sent us the sequence of what would become known as SARS-CoV-2. Two days later, Moderna allege that they developed a vaccine from this sequence within 48 hours of receiving it.
MUST WATCH! PATENT UNDERWRITER FOLLOWS THE COVID CRIME - CURE PATENTED BEFORE DISEASE This is an extremely important discussion that completely blows the lid off the covid scamdemic. This information must be shared everywhere and viewed by as many …
www.bitchute.com
Ralph Baric taught Shi Zhengli all his tricks of the trade. Peter Daszak funneled money to the WIV. They conducted gain-of-function research with federal funds during a federal funding moratorium, between 2014 and 2017.
Anthony Fauci lied in front of Congress about all of it, a felony.
Anthony Fauci, Shi Zhengli, Peter Daszak, and Ralph Baric are complicit in fraud, racketeering, and mass murder on an unimaginable scale.
It's high time we locked these motherfuckers all up and threw away the fucking key.
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