I found a vid about the Vagus Nerve (the 10th cranial nerve pair):
LOL, we
literally Deus Ex, now.
Basically, the pathway for the virus into the brain looks something like Lung Tissue > Parasympathetic Nerves of the Lungs > Vagus Nerve (CN X) > Botzinger Complex.
en.wikipedia.org
There are papers to support this conclusion:
The neuroinvasive potential of SARS‐CoV2 may play a role in the respiratory failure of COVID‐19 patients (2020-02-24) [https://onlinelibrary.wiley.com/doi/pdf/10.1002/jmv.25728]
In other words, the virus climbs the nerves of the lungs, then the nerves of the neck, and then right into the cardiorespiratory center of the brain stem. This happens to basically all of the critical patients. They suffer dysautonomia.
en.wikipedia.org
en.wikipedia.org
en.wikipedia.org
The autonomic nervous system controls all the parts of the body that you don't have any conscious control over, like breathing in your sleep, dilation of your pupils, peristalsis of the intestines, heartbeat, sexual arousal,
et cetera. Because the virus is a neurotropic respiratory virus, it gets into the nerves that control the lungs and from there, it rises all the way into
the part of the motherfucking medulla oblongata that controls your lungs.
Do people understand why this is so hard to diagnose? It's because these people already have breathing trouble from pneumonia. The neurological damage is masked by the direct cardiopulmonary damage.
With someone who is already having shortness of breath and pneumonia with obvious lung parenchymal damage, hardly anyone would suspect that the fucking cranial nerves and brain stem are contributing to their breathing troubles.
But it happens in transgenic hACE2 mice when they shoot SARS virus up their nose like fucking Flonase.
What this paper essentially says is that the virus gets into the mouse's medulla, homes in on the cardiorespiratory center, and causes aspiration pneumonia due to shallow breathing. The shallow breathing is, itself, caused by the nerves of the lungs being cut off from the brain by the virus directly damaging them.
Because this is a neurotropic virus.
I don't know how many times I have to say it before it sinks in. I've been shouting it from the rooftops basically fucking everywhere. Nobody wants to believe it.
Neurons have ACE2 receptors. This virus is more than capable of entering nerve cells and using them to replicate, as well as migrating through the nerves and into the brain.
More and more information on the neurological manifestations of this virus keeps coming out:
An abstract is unavailable.
journals.lww.com
For the love of god, stay indoors. Go full NEET if you have to. Just don't go out. Just stay the fuck home.
Our governments can't do it alone, we have to take action now! Do your part and stay the fuck home!
staythefuckhome.com
The economy doesn't matter. Wagecucking is bullshit anyway. Just don't get this goddamn virus.
Well, the data on chronic illness isn't in for COVID-19, yet. However, the syndrome that it causes is very,
very similar to SARS, and we have
lots of data on what SARS did to survivors.
To put it bluntly, it royally fucked them right the fuck up. They had lung scarring, chronic fatigue, mental issues, chronic pain, and osteonecrotic joint damage from being dosed with methylpred.
COVID-19 is similar enough to SARS that I am about 85% confident that this thing is going to leave behind shitloads of cripples and vegetables if we don't fucking do something about it right the fuck now.
Permanent. Functional. Impairment.