Wuhan Coronavirus - COVID-19 Analysis & Summary - This is not just fucking pneumonia. It is everything but the kitchen sink. Lungs, heart, kidneys, liver, brain, blood vessels, testes. It affects them all.

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Here David. I made this in MSpaint.
1586366373575.png

Red is fitted curve, green is the datapoints.
This model has an R-sq of 99%!!1! Wow, it's good isn't it? It must be generated with a formula!
No, if you look closely, you can see that the curve consistently underestimates and over estimates, you can see a very clear pattern.
Conclusion: Even if the R-sq is very high, the model could be bad.

You should now understand how R-sq actually works now.

Some might ask, why do I bother?
Simple, I'm bored out of my fucking mind, and I really dislike misinformation.
Combine that with being smug and condescending, it's asking me counter the misinformation.

I hate the liars of People's Daily as much as these unreliable shitty misleading "journalism".

His nickname should be "Drain Clogger" because he perpetually shits himself.
Train Clopping Clogger, sounds about right for a brony.
 
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The refutations are there, champ, you're just too obsessive to see them. You're breathlessly repeating worst-case scenarios, which are shared by whole families of viral infections, as if they're going to happen to everyone who so much as looks at this virus from across the room.

Ahh, there it is. The only counter-argument with even the barest hint of substance.

"But COVID-19 shares all these features with Influenza, which can also cause neurological and multi-organ complications, this is nothing new."

Please explain what sort of influenza causes people to collapse and seize in the street:













Encephalitis with influenza is rare. Very rare. In H1N1, the rate is like 12 per million people:



COVID-19 seems to cause encephalitis left and right, if these leaked videos are anything to go by.

No I didn't.
Division/multiplication first, then plus.
View attachment 1220387
So it's that first number plus 1. You left out the brackets.

Like I said, I copied and pasted it out of Windows Calculator. It had that order of operations already.

I did think about bracketing it just to be sure, but I thought it was fairly unambiguous what I actually meant based on the sum, so I didn't bother.

Of course their data is dishonest. Corruption will exist on many levels, from hospital, to different levels in the state. Hospital takes out 1 or 2 to make themselves look nice, then the politicians alter the sources more. Results in astronomical data errors.
Already 2 high level politicians have been arrested for covering up the data to save their role.
So what, doesn't mean the data is bad. It's an inherent flaw in sampling.

Let's see, the article you dug up starts out with China's economical data which we all know is unreliable.

So this "quantitative-finance" specialist fit a regression curve for the data, and obtained a high r-sq?
R-sq is almost meaningless in this context. R-sq only shows how much explanatory power the model has in terms of variance, for the regression line.
They didn't justify why they used cumulative deaths instead of day-to-day deaths.
99.99% means that 99.99% variance can be explained with the formula they made. That's it. A model with 99.99% R-sq can be a shit model.

You don't know what R-sq is. R-sq can be increased by simply including larger data. If I want to make a model to predict a person's location on the autism spectrum, I can include data such as the amount of posts on Kiwifarms, their height, weight, favourite colour, race, times they've had sex, number of dildos they own, number of pony plushies.... you fucking get it.
R-sq isn't dictatorial.
This is what we call "chance correlations", spurious data is bad.
View attachment 1220717
Oh no, spending money on space causes people to hand themselves! Nicolas Cage appearing in films cause people to fall into their pools and drown!

This is what R-sq does:
- Explains variability of the model
- Fitness of the model
- Layman's terms "how close is the dot to the line? Closer, better R-sq"

This is what R-sq can't do:
- Explain bias
- Explain the reliability of the model

The article you linked suck because:
- No graphs, I'll make my own
- Is the data normal?
- Residual plots? Just because R-sq is high, doesn't mean there's a good random spread of data.
- Is the adjusted R-sq 99.99%?


Agreed, as a statistician, it also makes me question the model.
The biggest problem with their number is the data they used. Presumably it's WHO's update reports. This alone makes their analysis stupid.
- Under China, Hong Kong, Macau, and Taiwan is included
- This suggests that HK and TW are in with the CPC, forging numbers, definitely not the case
- Maybe China accounted for that? Sure, China's able to predict the growth, right? No they can't. So this suggests that China and WHO are together, making fake data
- 99% r-sq doesn't mean the data is fake. Maybe their model sucks gigantic dick, maybe they don't know what they are talking about.
View attachment 1220690
I'm gathering WHO's data now, maybe you shouldn't skip your statistics class. All this shit is taught in a first year's statistics class, and you're failing so hard.

You can send me $500 for this course to bateatingchong@cpc.gov.cn, via Alipay.

Okay, I'll grant that's a good point.

But if that is the case, then why were people pushing the lack of variance in the data like it was some kind of smoking gun? Mind you, it wasn't just Barron's saying this. There were numerous people trying to claim that China's data fit a curve a little too perfectly.

This is what the news is trying to say over here:





This is quickly devolving into a shitstorm of opprobrium and international name-calling. I am honestly worried about the geopolitical ramifications of all of this.

And now, I'm seeing claims that lifting lockdowns is going to lead to a resurgence in cases:



Is it really believable that there are no COVID-19 deaths occurring over there right now?



So? You're regurgitating shit we all know.
The deaths are well under-estimated. We all know it.
CPC lies, or 2+2=4. Wow I am surprised.
If someone dies of Coronachan and wasn't previously diagnosed, they aren't included.
No diagnosis, not included in dataset, not verified to carry the virus.
Also for a city like Wuhan, there's people dying naturally everyday anyway.
This applies to every country, not just China.
A very low percentage of the population was tested, out of this small sample, some are dead.
This also tells me that you really don't know what you're talking about. As time goes on, people die of causes other than the disease. Depression from being indoors, ageing, dying from cancer because hospitals are full, suicide after reading this exceptional post of yours... All this will skew the CFR you're desperately trying to push.
Doesn't mean I think China's numbers are accurate, they definitely are not.



Did you just calculate CFR by dividing death with death plus recovery? ( Also, 17127/(17127+24392)=41.25099...%, or rounded to 41.3%... but oh well. C- for effort)
(Null should let us use LATEX so I can sperg more)
You still don't understand. The formula you're using is only applicable after the epidemic.

135586 cases, 17127 deaths, 24392 recovered.
CFR: 17127/135586=12.6318%
RCFR is that 41.3% of yours.
Still that CFR is 6 times higher than overall for China, but so what?
Why are you comparing the RCFR of China where the virus is effectively over to Italy, where coronachan is still circulating, looking for the next pizza chef to infect?
Didn't I point out how wrong it was in my initial response?

China locked down Wuhan, then the province, then the surrounding provinces in less than a week from the 23rd.
Italy let the virus spread.
China could have done all this 7-10 days earlier, if the politicians of Wuhan weren't corrupted traitors of the nation that tried to cover the virus up.
Italy could have done this... on the first day of fucking March.

Maybe, just maybe Italy sucks.

View attachment 1220594
South Korea is claiming 6776 recoveries and 200 deaths.
RCFR: 200/6976=2.8670%
CFR: 200/10384=1.9260% Diagnosed, 2% death rate.

View attachment 1220600
RCFR: 6/412=1.4563%
CFR: 6/1623=tiny

View attachment 1220672
RCFR: 50/1086=4.6041%
CFR: 50/6010=<1%

Italy sucks.
China's numbers are fake.
But Italy still sucks.

You know, I find it uncanny that when you take China's claimed 77,000 recoveries and assume about 50,000ish deaths (as the whole urns situation is suggesting), the RCFR is very, very similar to Italy's current RCFR. About 1 in 2.5 of known cases died.

We are assuming there are about 80,000 cases in China, right? Well, lockdowns have absolutely no positive effect on the mortality of existing infections. In fact, they can make it harder for people to access medical care.

Why is the ratio of recovered cases to deaths ten times better in China than in Italy?

@Drain Todger

Has any Britfag ever pointed out that by switching letters in your original nickname, you actually gave yourself a nickname that amounts to "trash dick"?

They don't have to. I was aware of that when I made it. I thought it was funny.

Here David. I made this in MSpaint.
View attachment 1220773
Red is fitted curve, green is the datapoints.
This model has an R-sq of 99%!!1! Wow, it's good isn't it? It must be generated with a formula!
No, if you look closely, you can see that the curve consistently underestimates and over estimates, you can see a very clear pattern.
Conclusion: Even if the R-sq is very high, the model could be bad.

You should now understand how R-sq actually works now.

But there isn't even that much variance in the data that I saw. It wasn't just Barron's that made this claim that the data was fabricated to fit a curve, you know:





This stuff did the rounds everywhere.
 
Ahh, there it is. The only counter-argument with even the barest hint of substance.

"But COVID-19 shares all these features with Influenza, which can also cause neurological and multi-organ complications, this is nothing new."

Please explain what sort of influenza causes people to collapse and seize in the street:













Encephalitis with influenza is rare. Very rare. In H1N1, the rate is like 12 per million people:



COVID-19 seems to cause encephalitis left and right, if these leaked videos are anything to go by.



Like I said, I copied and pasted it out of Windows Calculator. It had that order of operations already.

I did think about bracketing it just to be sure, but I thought it was fairly unambiguous what I actually meant based on the sum, so I didn't bother.



Okay, I'll grant that's a good point.

But if that is the case, then why were people pushing the lack of variance in the data like it was some kind of smoking gun? Mind you, it wasn't just Barron's saying this. There were numerous people trying to claim that China's data fit a curve a little too perfectly.

This is what the news is trying to say over here:





This is quickly devolving into a shitstorm of opprobrium and international name-calling. I am honestly worried about the geopolitical ramifications of all of this.

And now, I'm seeing claims that lifting lockdowns is going to lead to a resurgence in cases:



Is it really believable that there are no COVID-19 deaths occurring over there right now?





You know, I find it uncanny that when you take China's claimed 77,000 recoveries and assume about 50,000ish deaths (as the whole urns situation is suggesting), the RCFR is very, very similar to Italy's current RCFR. About 1 in 2.5 of known cases died.

We are assuming there are about 80,000 cases in China, right? Well, lockdowns have absolutely no positive effect on the mortality of existing infections. In fact, they can make it harder for people to access medical care.

Why is the ratio of recovered cases to deaths ten times better in China than in Italy?



They don't have to. I was aware of that when I made it. I thought it was funny.



But there isn't even that much variance in the data that I saw. It wasn't just Barron's that made this claim that the data was fabricated to fit a curve, you know:





This stuff did the rounds everywhere.
Nigga they're just doing the Harlem shake
 
Ahh, there it is. The only counter-argument with even the barest hint of substance.

"But COVID-19 shares all these features with Influenza, which can also cause neurological and multi-organ complications, this is nothing new."

Please explain what sort of influenza causes people to collapse and seize in the street:













Encephalitis with influenza is rare. Very rare. In H1N1, the rate is like 12 per million people:



COVID-19 seems to cause encephalitis left and right, if these leaked videos are anything to go by.



Like I said, I copied and pasted it out of Windows Calculator. It had that order of operations already.

I did think about bracketing it just to be sure, but I thought it was fairly unambiguous what I actually meant based on the sum, so I didn't bother.



Okay, I'll grant that's a good point.

But if that is the case, then why were people pushing the lack of variance in the data like it was some kind of smoking gun? Mind you, it wasn't just Barron's saying this. There were numerous people trying to claim that China's data fit a curve a little too perfectly.

This is what the news is trying to say over here:





This is quickly devolving into a shitstorm of opprobrium and international name-calling. I am honestly worried about the geopolitical ramifications of all of this.

And now, I'm seeing claims that lifting lockdowns is going to lead to a resurgence in cases:



Is it really believable that there are no COVID-19 deaths occurring over there right now?





You know, I find it uncanny that when you take China's claimed 77,000 recoveries and assume about 50,000ish deaths (as the whole urns situation is suggesting), the RCFR is very, very similar to Italy's current RCFR. About 1 in 2.5 of known cases died.

We are assuming there are about 80,000 cases in China, right? Well, lockdowns have absolutely no positive effect on the mortality of existing infections. In fact, they can make it harder for people to access medical care.

Why is the ratio of recovered cases to deaths ten times better in China than in Italy?



They don't have to. I was aware of that when I made it. I thought it was funny.



But there isn't even that much variance in the data that I saw. It wasn't just Barron's that made this claim that the data was fabricated to fit a curve, you know:





This stuff did the rounds everywhere.
Literally quoting people to posit your conspiratard takes.
And you still wonder why refuting you instead of your arguments is better.
 
Ahh, there it is. The only counter-argument with even the barest hint of substance.

"But COVID-19 shares all these features with Influenza, which can also cause neurological and multi-organ complications, this is nothing new."

Please explain what sort of influenza causes people to collapse and seize in the street:













Encephalitis with influenza is rare. Very rare. In H1N1, the rate is like 12 per million people:



COVID-19 seems to cause encephalitis left and right, if these leaked videos are anything to go by.



Like I said, I copied and pasted it out of Windows Calculator. It had that order of operations already.

I did think about bracketing it just to be sure, but I thought it was fairly unambiguous what I actually meant based on the sum, so I didn't bother.



Okay, I'll grant that's a good point.

But if that is the case, then why were people pushing the lack of variance in the data like it was some kind of smoking gun? Mind you, it wasn't just Barron's saying this. There were numerous people trying to claim that China's data fit a curve a little too perfectly.

This is what the news is trying to say over here:





This is quickly devolving into a shitstorm of opprobrium and international name-calling. I am honestly worried about the geopolitical ramifications of all of this.

And now, I'm seeing claims that lifting lockdowns is going to lead to a resurgence in cases:



Is it really believable that there are no COVID-19 deaths occurring over there right now?





You know, I find it uncanny that when you take China's claimed 77,000 recoveries and assume about 50,000ish deaths (as the whole urns situation is suggesting), the RCFR is very, very similar to Italy's current RCFR. About 1 in 2.5 of known cases died.

We are assuming there are about 80,000 cases in China, right? Well, lockdowns have absolutely no positive effect on the mortality of existing infections. In fact, they can make it harder for people to access medical care.

Why is the ratio of recovered cases to deaths ten times better in China than in Italy?



They don't have to. I was aware of that when I made it. I thought it was funny.



But there isn't even that much variance in the data that I saw. It wasn't just Barron's that made this claim that the data was fabricated to fit a curve, you know:





This stuff did the rounds everywhere.
Dave pls.
 
Word salad

lolcalmdown

I though this kind of faggotry was mostly for under developed teenagers trying to win pointless internet arguments but here we have a grown man throwing around walls of text with unrelated links all over the place.

Brevity is the soul of wit David.

Division/multiplication first, then plus.
View attachment 1220387
So it's that first number plus 1. You left out the brackets.

:story:

Holy shit. No comprehension of basic mathematics and he thinks he can lecture people about scientific papers on a complex subject like this.
 
COVID-19 seems to cause encephalitis left and right, if these leaked videos are anything to go by.
"If these totally unverified videos are legit, then COVID-19 could maybe possibly cause encephalitis"
You aren't even making an argument here
Is it really believable that there are no COVID-19 deaths occurring over there right now?
Of course it's not believable you illiterate autist.
You know, I find it uncanny that when you take China's claimed 77,000 recoveries and assume about 50,000ish deaths (as the whole urns situation is suggesting), the RCFR is very, very similar to Italy's current RCFR. About 1 in 2.5 of known cases died.

We are assuming there are about 80,000 cases in China, right? Well, lockdowns have absolutely no positive effect on the mortality of existing infections. In fact, they can make it harder for people to access medical care.

Why is the ratio of recovered cases to deaths ten times better in China than in Italy?
Asking questions like that doesn't make you look smart, it actually does the exact opposite. You don't have any answers, so you're copying what your special ed teacher does when she wants to lead you towards the right answer. It's painfully transparent and you're not fooling anyone.
 
Nigga they're just doing the Harlem shake

But we now have multiple case studies showing that the virus was found in cerebrospinal fluid and brain tissue. Mind you, SARS was known to be neurotropic, as well. Not only was it found in the brains of victims of the 2003 outbreak, it was also found to be able to infect the olfactory bulbs of transgenic hACE2 mice. There's tons of evidence for it. ACE2 receptors are indeed found in human neurons and the virus can attack those tissues and infect them directly.

Evidence of the COVID-19 Virus Targeting the CNS: Tissue Distribution, Host–Virus Interaction, and Proposed Neurotropic Mechanisms (2020-03-13) [https://pubs.acs.org/doi/10.1021/acschemneuro.0c00122]

The brain has been reported to express ACE2 receptors (Figure 1A, C) that have been detected over glial cells and neurons, which makes them a potential target of COVID-19. Previous studies have shown the ability of SARS-CoV to cause neuronal death in mice by invading the brain via the nose close to the olfactory epithelium.(3) The contribution of the neurotropic potential of SARS-CoV-2 in patients reported in the recent outbreak of COVID-19 remains to be established. In the SARS-CoV infections that were reported in the past, autopsy findings of the patients have shown strong evidence of the presence of SARS-CoV by electron microscopy, immunohistochemistry, and real-time reverse transcription-PCR (3). Patients with acute SARS-CoV illness have also demonstrated the presence of the virus in cerebrospinal fluid. The role of the blood-brain barrier in containing the virus and preventing it from gaining access to the neural tissues needs to be further explored in patients diagnosed with COVID-19. Recently, a study posted in medRxiv(4) has reported neurological manifestations in COVID-19 in the current outbreak that involved 214 patients, of which 78 (36.4%) patients had neurologic manifestations, which affirms our rationale of the neurotropic potential in the COVID-19 virus. Also, a finding published on a patient who had loss of involuntary control over breathing(5) during the recent outbreak with several other patients suffering acute respiratory failure implores healthcare professionals and clinicians to segregate COVID-19 patients into neurologically affected cases and those who are devoid of neurological deficits.

You know what other tissues in the body have ACE2 receptors? The endothelial lining of the blood vessels and the blood-brain barrier.



What if the virus can break through the BBB and into the brain by direct endothelial cell infection? How would we even know? Have they even looked for evidence of this?



There is lots and lots of evidence for the highly aggressive neurotropism of this virus. People are suffering anosmia, the loss of their sense of smell. How do you know if that's from an infection of their olfactory nerve or not?

Literally quoting people to posit your conspiratard takes.
And you still wonder why refuting you instead of your arguments is better.

I know what I saw.






I know people are trying to cope with gallows humor, and in ordinary circumstances, I would be right there with you, laughing at the absurdity of all this.

However, I don't happen to think that any of this is very fucking funny at all.

It's actually not particularly funny that a highly contagious neurotropic airborne virus is going around that can, in some cases, make people collapse on the ground with seizures and die.

It is hilarious, however, that libtards who would ordinarily never even entertain the idea that such a thing as crisis actors exist have come out against these videos claiming that they're fake and full of crisis actors.

It's especially hilarious when even the fucking Guardian thinks the videos are real:



After President Xi Jinping ordered “resolute efforts” to curb the spread of coronavirus in his first public remarks on the disease on 20 January, Wuhan was swiftly placed under lockdown. Millions of communities across China also began to implement draconian epidemic control measures.

The rough implementation of epidemic control has resulted in extensive human rights abuses across China, analysts say. Disturbing images have emerged on social media showing people chained up and paraded on streets, or beaten by police for not wearing masks. Footage has also shown officers installing metal bars or chains outside people’s homes to prevent them leaving.

Propaganda banners with threatening messages have been put up around the country. “A surgical mask or a breathing tube? Your choice,” said one. “Those who don’t report their fever are class enemies hiding among the people,” another said.

Now you're a "conspiratard" for agreeing with a position held by the fucking Guardian.

My fucking sides.

Why won't you answer my questions, David? I'm asking things you can actually answer, not questions better put to someone with relevant education.

Maybe because it's a red herring and irrelevant to this discussion?
 
ACE2 RECEPTORS ON OTHER ORGANS ARE IMPORTANT BECAUSE I SAY SO, BUT WE DON'T EVEN KNOW IF IT BREAKS THE BBB YET.
Academics pls respond.

Oh also speaking of psych profiles I noticed you fall back on spastically citing retarded youtube videos when you're stressed in an attempt to drown out dissenting opinion, while sprinkling what amounts to a piddly non distributio medii (oh hey that's why your retardo latin is itallicised, lol)

Good job totally not caring tho.
 
@Drain Todger you're mistaking absolute numbers for relative. Again. The sheer number of infections in such a short time would mean that of course a large number of people would suffer extreme symptoms on that same short period, but that's a meaningless number. If half the planet caught flu at the same time, it would also result in an abundance of extreme reactions in a relatively short period simply because of the numbers involved.

Again, you are utterly shit at assessing rekative risks and have no concept of small Vs far away.
 
Here David. I made this in MSpaint.
View attachment 1220773
Red is fitted curve, green is the datapoints.
This model has an R-sq of 99%!!1! Wow, it's good isn't it? It must be generated with a formula!
No, if you look closely, you can see that the curve consistently underestimates and over estimates, you can see a very clear pattern.
Conclusion: Even if the R-sq is very high, the model could be bad.

You should now understand how R-sq actually works now.

Some might ask, why do I bother?
Simple, I'm bored out of my fucking mind, and I really dislike misinformation.
Combine that with being smug and condescending, it's asking me counter the misinformation.

I hate the liars of People's Daily as much as these unreliable shitty misleading "journalism".


Train Clopping Clogger, sounds about right for a brony.
OH NOES COSINE FUNCTIONS ARE CORRELATING WITH THE DIFFERENTIALS ALMOST PERFECTLY, THAT NO MAKE NO SENSE MAMA MIA!

Nigga they're just doing the Harlem shake
Guess these are COVID cases as well, fuck guys we're all beat!

Academics pls respond.

Oh also speaking of psych profiles I noticed you fall back on spastically citing exceptional youtube videos when you're stressed in an attempt to drown out dissenting opinion, while sprinkling what amounts to a piddly non distributio medii (oh hey that's why your retardo latin is itallicised, lol)

Good job totally not caring tho.
ACE-2 Receptors are important in how the virus binds in the lungs. Initially it is believed that was the primary cause, and only factor in relation to the COVID deaths. There is data supporting the hemoglobin levels getting jammed up physically by the virus binding to the Iron ions in the blood, giving COVID patients more along the lines of high altitude sickness, and the pneumonia is the later stage effect once the virus has totally taken control. The primary driver initially is beginning to get proven to be an side effect for the actual underlying prognosis, which makes sense for a NOVEL viral pathogen. Earlier testing and being able to shorten the differential between complications for the unlucky 5% is the biggest issue in medicine currently, this type of aliment hasn't popped up in almost a century. Luckily the global community thanks to modern science is able to tackle this problem faster than in 1918 unlike what the media likes people to think.
 
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Academics pls respond.

Yeah. Let's get some actual academics in this thread who understand biology.

Mind you, this virus doesn't actually have to get through the BBB at all, because it can ascend the parasympathetic nerves of the lungs, enter the vagus nerve, and then enter the brain stem.

Influenza can pull off this neat little trick, too:



The difference is that with Influenza, it's really, really rare. With SARS-CoV-2, it's common, because this virus deeply invades various tissues, and it does so reliably.

Oh, by the way, I contacted actual PhDs, including Dr. Todd Rider and Dr. Kevin McCairn, and not only did they agree with my conclusions, they gave my work the thumbs-up.


This is the same guy who said my video on the virus would "put most MDs and PhDs to shame".

gdIhqpW.jpg


Legit, actual scientist:


Calipers permanently BTFO.

@Drain Todger you're mistaking absolute numbers for relative. Again. The sheer number of infections in such a short time would mean that of course a large number of people would suffer extreme symptoms on that same short period, but that's a meaningless number. If half the planet caught flu at the same time, it would also result in an abundance of extreme reactions in a relatively short period simply because of the numbers involved.

Again, you are utterly shit at assessing rekative risks and have no concept of small Vs far away.

The surge of cases is dangerous because it causes deaths-by-proxy. In other words, if hospitals are suddenly jammed full of COVID-19 patients, they'll have a harder time treating cancer, strokes, heart attacks, gunshot wounds... all other causes of death. If a hospital is suddenly overwhelmed, it increases mortality from all causes. There's also the extremely high risk of nosocomial infections with this virus. If you introduce COVID-19 patients into a hospital, guess what? Everyone else who is being treated there will start getting sick with it as well, simply due to being in the same building with them.

One of the earliest reports on the virus indicated that 41 fucking percent of the patients they counted in the study were hospital-acquired infections.



Oh, look. They're digging mass graves in New York, now.

 
ACE-2 Receptors are important in how the virus binds in the lungs. Initially it is believed that was the primary cause, and only factor in relation to the COVID deaths. There is data supporting the hemoglobin levels getting jammed up physically by the virus binding to the Iron ions in the blood, giving COVID patients more along the lines of high altitude sickness, and the pneumonia is the later stage effect once the virus has totally taken control. The primary driver initially is beginning to get proven to be an side effect for the actual underlying prognosis, which makes sense for a NOVEL viral pathogen. Earlier testing and being able to shorten the differential between complications for the unlucky 5% is the biggest issue in medicine currently, this type of aliment hasn't popped up in almost a century. Luckily the global community thanks to modern science is able to tackle this problem faster than in 1918 unlike what the media likes people to think.
The problem is that he thinks ACE-2 receptors on other organs matter, and so far have been citing the SAME study only saying that the brain has those receptors instead of explaining why it's relevant in the first place, while somehow knowing the Blood-brain barrier exists.

It's like he's trying to argue while trying to get us to make his arguments for him. It's pretty retarded.

Dude makes a hypothesis and ASKS NORMIES TO DO THE RESEARCH FOR HIM.

gdIhqpW.jpg


Legit, actual scientist:

GG on using a neurologist's word as proof of virology ethos btw, that's such a good take.

lul.

You really need new material.
How about try citing Black Science Man next time?
 
ACE-2 Receptors are important in how the virus binds in the lungs. Initially it is believed that was the primary cause, and only factor in relation to the COVID deaths. There is data supporting the hemoglobin levels getting jammed up physically by the virus binding to the Iron ions in the blood, giving COVID patients more along the lines of high altitude sickness, and the pneumonia is the later stage effect once the virus has totally taken control. The primary driver initially is beginning to get proven to be an side effect for the actual underlying prognosis, which makes sense for a NOVEL viral pathogen. Earlier testing and being able to shorten the differential between complications for the unlucky 5% is the biggest issue in medicine currently, this type of aliment hasn't popped up in almost a century. Luckily the global community thanks to modern science is able to tackle this problem faster than in 1918 unlike what the media likes people to think. EDIT:(EVEN IF THE LUNGS AREN'T THE ONLY REASON, IT STILL CAN BE ASSUMED THAT MOST LITERATURE STATES THE PROMIENCE OF ACE-2 RECPITORS FOR BINDING IN THE LUNGS IN EARLY STUDIES/OBSERVATIONS MADE OUTSIDE OF STUDIES. THE FUNCTIONALITY IN ALL ORGANS IS WIDELY MISUNDERSTOOD, BUT STILL MY SUCCEDDING POINTS STAND.)
 
Autistic drivel
1. I'm in the middle of getting the data ready. Going to ignore all the babble coming from economists.
2. Of course it's not believable that no one is dying from the virus in China, because:
1586368725148.png

Notice how China's numbers are very similar to Korea's?
Oh and yeah, people are still dropping dead from Coronachan.
3. China's intelligence claims US citizens hunt nigggers for fun (I condone hunting bronies and furries for fun), USA intelligence claims China has fake numbers... yawn. I have acknowledged more than twice that the numbers are definitely underestimated.
4. Lockdown and authoritarian fist makes people scared to go out and pass the virus around. Of course there's existing conditions. China could have responded 7 days earlier if those Wuhan politicians weren't dipshits, and the numbers would be even more mundane looking. This still doesn't mean the virus won't blow up elsewhere.
Funny how you simply ignore South Korea and Singapore, both of which have better numbers than China. So are they lying too?
5. Thanks for that twitter. It's going to be pretty useful when I analyse the data. Important information is still missing, a child can fit a curve over some data. His predictions became very unreliable after mid-Feb.
 
That’s my whole point. You habitually indulge in ridicule because rhetoric simply escapes you. You people do not realize that ridicule is not a substitute for rhetoric, nor does it augment rhetoric in any way, shape, or form.

This is not a problem specific to Kiwi Farms. It is common for mouth-breathers on the internet to confuse contradiction with argument. This happened on SpaceBattles all the time. Nine times out of ten, nobody actually bothers to refute anything. All they do is invoke the burden of proof and demand more and more citations, and then, when you do actually cite your sources, they say that your sources mean the opposite of what you think. It is argumentation by means of Kafkaesque gaslighting, and it is not valid.

COVID-19 causes severe autoimmune damage in multiple vital organs, and it causes sepsis and widespread clotting that eventually leads to infarcts. All of these patients have elevated inflammatory cytokines and elevated D-dimer levels indicative of this.



I have provided ample evidence of these claims. Now, if you’re going to argue this, you have to actually refute the fucking claim.

“Oh, that’s crazy, that’s absurd, you’re being silly, let's discuss how you came to be so silly and ignore the content of your argument,” is not a refutation. Trust me, you Kiwis aren’t the only ones to get that wrong. I see this sort of faux-argumentation everywhere online, where all argument consists essentially of attacking credibility of sources or simply declaring ideas absurd without any actual refutation. It is an anti-intellectual poison that is ruining online discourse, and I tire of it.

Stop engaging in Bulverism and refute the fucking claim.
You have autism.
 
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